Case 1. M.S., a railroad-car inspector, was first seen at the White River Junction Veterans Administration Hospital in 1941, at 43 years of age, complaining of "lumbago" and sensitivity of the ...

The bronze-like diabetes recognized in the early 20th century is now a rare "tip of the iceberg" manifestation of HH. High clinical suspicion is a prerequisite in the detection of HH cases. This ...

Hemochromatosis is a disorder that causes your body to absorb more iron than it should, a condition called iron overload. It affects about one million people in the United States, making it one of the ...

LIVER tumors, usually hepatomas, are relatively frequent in cases of hepatic cirrhosis, occurring in approximately 3 or 4 per cent of patients with alcoholic cirrhosis and in a somewhat higher ...

Hereditary hemochromatosis is a genetic condition that causes the body to absorb too much dietary iron, usually in adulthood. Symptoms can include tiredness, weight loss, skin color changes, and more.

Haemochromatosis causes a build-up of iron in the body which can cause harm to joints and organs – although the extent of this harm is unclear, especially in older ages. The new research, led by a ...

Nat Clin Pract Gastroenterol Hepatol. 2006;3(4):178-179. Although hemochromatosis was first described in the 19 th century, John Sheldon's 1934 description of 300 cases remains a landmark publication ...

Hemochromatosis causes the body to absorb too much iron from foods. To minimize the symptoms and reduce the risk of complications, people can follow a hemochromatotis diet. There are two types of ...

Hemochromatosis is a disorder that causes your body to absorb too much iron. Blood tests that measure iron levels in your blood and liver can help diagnose the condition. The medical disorder ...

Thousands of people globally with an iron disorder have their blood regularly thrown out—Australian research shows it can be used to save lives Many countries regularly experience shortages of blood ...

Although hemochromatosis was first described in the 19 th century, John Sheldon's 1934 description of 300 cases remains a landmark publication in defining the clinical characteristics of the disease.